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NEW QUESTION: 1
. You are designing an n-tier Windows application by using Microsoft .NET Framework 4, Microsoft Visual Studio 2010, and Microsoft SQL Server 2008. The application will replace an existing client/server application. The existing application was created by using Microsoft Visual Basic 6.0 and consists of a series of COM components that access a SQL Server database. You plan to move the existing COM components into a Windows Communication Foundation (WCF) tier. You need to ensure that the COM components can be replaced without impacting the existing user interface (UI) tier. You also need to ensure that the COM components can be replaced separately. What should you do?
A. Create a common assembly on the WCF tier of the new application to interface with the COM components.
B. Use .NET COM Interop on the client tier to interact directly with the COM components until they are replaced by the managed code. http://www.test4pass.com Leading the way in IT Certification Exams Test4pass Expert In IT Certification Exams
C. Create a common assembly on the UI tier of the new application to interface with the COM components
D. Convert the Visual Basic 6.0 source code to managed code by using the Visual Studio converters. Use these code components on the client/server application until they are replaced individually with the new permanent managed code functionality.
Answer: A

NEW QUESTION: 2
Refer to the exhibit.

Assume that Student A's ARP table is empty. Student A prepares to send an FTP packet to FTP Server B and needs to discover the proper destination MAC address.
For which IP address does Student A send an ARP request?
A. 10.1.20.1
B. 10.1.10.87
C. 10.1.10.1
D. 10.1.2.2
Answer: D
Explanation:
Explanation/Reference:

NEW QUESTION: 3
Identify the environment variable that points to the $COMMON_TOP/java directory in Oracle Applications R12.
A. JAVA_BASE
B. JRE_TOP
C. JDK_TOP
D. JAVA_TOP
Answer: A

NEW QUESTION: 4
Select the class of Anti-diabetic medication that works in the specified organ to prevent hyperglycemia. Select all that applies. Kidney (G)
A. Alpha- Glucosidase Inhibitors
B. Thiazolidinediones
C. Biguanide
D. DPP4 Inhibitors
E. Sulfonylureas
F. Glucagon-like peptide-1 receptor agonists
G. SGLT2 inhibitors
Answer: G
Explanation:
Explanation
SGLT2 inhibitors Sulfonylureas work in beta cells in the pancreas that are still functioning to enhance insulin secretion. Alpha-Glucosidase Inhibitors stop -glucosidase enzymes in the small intestine and delay digestion and absorption of starch and disaccharides which lowers the levels of glucose after meals. DPP4 blocks the degradation ofGLP-1, GIP, and a variety of other peptides, including brain natriuretic peptide. Glucagon-like peptide-1 receptor agonists work in various organs of the body. Glucagon-like peptide-1 receptor agonists enhance glucose homeostasis through: (i) stimulation of insulin secretion; (ii) inhibition of glucagon secretion; (iii) direct and indirect suppression of endogenous glucose production; (iv) suppression of appetite; (v) enhanced insulin sensitivity secondary to weight loss; (vi) delayed gastric emptying, resulting in decreased postprandial hyperglycaemia. Thiazolidinediones are the only true insulin-sensitising agents, exerting their effects in skeletal and cardiac muscle, liver, and adipose tissue. It ameliorates insulin resistance, decreases visceral fat. Biguanides work in liver, muscle, adipose tissue via activation of AMP-activated protein kinase (AMPK) reduce hepatic glucose production. SGLT2 inhibitors work in the kidneys to inhibit sodium-glucose transport proteins to reabsorb glucose into the blood from muscle cells; overall this helps to improve insulin release from the beta cells of the pancreas.